Component
Supraventricular ectopy
Premature cardiac electrical events originating above the ventricles, detected by Holter monitoring.
2 recorded relationships. Experimental role, claim status and evidence remain attached to each record.
How nutrients influence it
Every nutrient with a recorded effect on this component, credited to the nutrient that acted rather than the chapter that recorded it. Open a nutrient to see the findings and the conditions they were measured under.
Other things that act on it
Enzymes, hormones, genes, and other components with a recorded effect. These are not nutrients, so they do not count toward the arrows above. Each finding names the chapter that recorded it.
How nutrients reach it in more than one step
Chains of two or more recorded steps that end here, grouped by the nutrient they start from. Each step is a separate finding, so a chain is a route a mechanism could take, not proof that it does.
Tracing routes…
What it does
Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.
What acts on it
Lower Mg intake increased supraventricular ectopy in a randomized crossover feeding study of postmenopausal women.
Experimental context and source evidence
- availability_state
- nutrient_deficiency Imported condition classification; unverified.
- evidence-system
- 22 completers; 81-day low and supplemented Mg periods; Holter monitoring
- experimental_model
- 22 completers; 81-day low and supplemented Mg periods; Holter monitoring
- exposure
- Chemically measured mean intake: 130 mg Mg/day in the low period versus 411 mg/day in the supplemented period; 81 days per period after 10-day equilibration. These are experimental exposures, not recommendations.
- limitations
- Ventricular ectopy alone was not significantly increased; small cohort. Dietary copper varied independently; reported Mg effect was unaffected by copper or sequence.
- nutrient_topic
- Magnesium research collection; topical membership is not evidence of a direct dietary effect. · Magnesium
- organism
- Human
- plain_language
- Controlled magnesium restriction produced its own measurable cardiac electrical effect.
- primary_references
- [klevay-2002-ectopy] Low dietary magnesium increases supraventricular ectopy (2002). https://ajcn.nutrition.org/article/S0002-9165%2823%2906154-3/fulltext DOI: 10.1093/ajcn/75.3.550
- tissue
- Whole-body nutrition; cardiac electrophysiology
- tissue_or_cell_type
- Whole-body nutrition; cardiac electrophysiology
- trigger_kind
- nutrient_deficiency Imported condition classification; unverified.
Magnesium: cross-nutrient mechanisms and deficiency (2026-09-17) · lines 1318–1330
AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · 22 completers; 81-day low and supplemented Mg periods; Holter monitoring · source_derived_draft · unverified_draft
### low-dietary-magnesium-increases-ectopy Lower Mg intake increased supraventricular ectopy in a randomized crossover feeding study of postmenopausal women. Condition category: nutrient_deficiency nutrient_topic: Magnesium research collection; topical membership is not evidence of a direct dietary effect. plain_language: Controlled magnesium restriction produced its own measurable cardiac electrical effect. organism: Human tissue_or_cell_type: Whole-body nutrition; cardiac electrophysiology experimental_model: 22 completers; 81-day low and supplemented Mg periods; Holter monitoring limitations: Ventricular ectopy alone was not significantly increased; small cohort. Dietary copper varied independently; reported Mg effect was unaffected by copper or sequence. exposure: Chemically measured mean intake: 130 mg Mg/day in the low period versus 411 mg/day in the supplemented period; 81 days per period after 10-day equilibration. These are experimental exposures, not recommendations. evidence-system: 22 completers; 81-day low and supplemented Mg periods; Holter monitoring tissue: Whole-body nutrition; cardiac electrophysiology [klevay-2002-ectopy] Low dietary magnesium increases supraventricular ectopy (2002). https://ajcn.nutrition.org/article/S0002-9165%2823%2906154-3/fulltext DOI: 10.1093/ajcn/75.3.550
Complete structured claim and evidence
Where it participates (unsigned role)
Mg biomarkers declined on the low-Mg diet, but hypomagnesemia, hypocalcemia and hypokalemia were not detected.
Experimental context and source evidence
- availability_state
- biomarker_context Imported condition classification; unverified.
- evidence-system
- Crossover feeding and biochemical monitoring
- experimental_model
- Crossover feeding and biochemical monitoring
- exposure
- Chemically measured mean intake: 130 mg Mg/day in the low period versus 411 mg/day in the supplemented period; 81 days per period after 10-day equilibration. These are experimental exposures, not recommendations.
- limitations
- A study-specific biomarker observation; not a diagnostic rule for all normal serum values.
- nutrient_topic
- Magnesium research collection; topical membership is not evidence of a direct dietary effect. · Magnesium
- organism
- Human
- plain_language
- A normal-range serum result did not rule out the diet-related electrical phenotype in these participants.
- primary_references
- [klevay-2002-ectopy] Low dietary magnesium increases supraventricular ectopy (2002). https://ajcn.nutrition.org/article/S0002-9165%2823%2906154-3/fulltext DOI: 10.1093/ajcn/75.3.550
- tissue
- Serum, erythrocytes and urine
- tissue_or_cell_type
- Serum, erythrocytes and urine
- trigger_kind
- biomarker_context Imported condition classification; unverified.
Magnesium: cross-nutrient mechanisms and deficiency (2026-09-17) · lines 1332–1344
AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · Crossover feeding and biochemical monitoring · source_derived_draft · unverified_draft
### low-magnesium-diet-without-hypomagnesemia Mg biomarkers declined on the low-Mg diet, but hypomagnesemia, hypocalcemia and hypokalemia were not detected. Condition category: biomarker_context nutrient_topic: Magnesium research collection; topical membership is not evidence of a direct dietary effect. plain_language: A normal-range serum result did not rule out the diet-related electrical phenotype in these participants. organism: Human tissue_or_cell_type: Serum, erythrocytes and urine experimental_model: Crossover feeding and biochemical monitoring limitations: A study-specific biomarker observation; not a diagnostic rule for all normal serum values. exposure: Chemically measured mean intake: 130 mg Mg/day in the low period versus 411 mg/day in the supplemented period; 81 days per period after 10-day equilibration. These are experimental exposures, not recommendations. evidence-system: Crossover feeding and biochemical monitoring tissue: Serum, erythrocytes and urine [klevay-2002-ectopy] Low dietary magnesium increases supraventricular ectopy (2002). https://ajcn.nutrition.org/article/S0002-9165%2823%2906154-3/fulltext DOI: 10.1093/ajcn/75.3.550
Complete structured claim and evidence
The events it takes part in
A mechanism often involves more than two components. These are the full events, with every participant and its role.
Situations it appears in
Low-supply and faulty-machinery situations recorded in the chapters where this component plays a part.
In the sources
Preserved passages that mention this component, quoted exactly. Open one to read it in context.
Open hypotheses
Proposed ideas that involve this component. They are labeled as hypotheses and do not change any recorded statement.
This is a research prototype built from draft material. It is not medical advice, and its statements still await verification against the original studies.