Component

Neutrophil chemotaxis

Neutrophil chemotaxis

2 recorded relationships. Experimental role, claim status and evidence remain attached to each record.

Read recorded mechanisms Open recursive mind map

How nutrients influence it

Every nutrient with a recorded effect on this component, credited to the nutrient that acted rather than the chapter that recorded it. Open a nutrient to see the findings and the conditions they were measured under.

How nutrients reach it in more than one step

Chains of two or more recorded steps that end here, grouped by the nutrient they start from. Each step is a separate finding, so a chain is a route a mechanism could take, not proof that it does.

Tracing routes…

What it does

Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.

Recorded relationships

What acts on it

  1. After KI 15 mg/kg/day for three days, peripheral neutrophil chemotaxis decreased in 15 healthy subjects.

    Potassium iodide → Neutrophil chemotaxis source_derived_draftungraded
    Experimental context and source evidence
    experimental_model
    Human exposure; modified Boyden-chamber assay.
    exposure_category
    Study-specific exposure, including pharmacological and in-vitro conditions; normal is the schema fallback outside the three availability categories.
    limitations
    Small mechanistic study; disease benefit was proposed rather than demonstrated.
    nutrient_topic
    Potassium iodide research collection; shared-anion and comparator studies are not all KI interventions. · Potassium iodide
    plain_language
    KI changed how neutrophils moved toward a signal.
    primary_references
    Potassium iodide inhibits neutrophil chemotaxis. · 1990 · https://pubmed.ncbi.nlm.nih.gov/1972841/

    Potassium iodide: thyroid and non-thyroid mechanisms, interactions and discovery questions (2026-09-18) · lines 280–286

    AI-assisted research curation; primary-study references, chemical references and label statements individually identified. Not publisher full text. · supports · Human exposure; modified Boyden-chamber assay. · source_derived_draft · unverified_draft

    ## ki-chemotaxis KI changed how neutrophils moved toward a signal. After KI 15 mg/kg/day for three days, peripheral neutrophil chemotaxis decreased in 15 healthy subjects. Model: Human exposure; modified Boyden-chamber assay. Limitations: Small mechanistic study; disease benefit was proposed rather than demonstrated. Evidence location: Primary abstract Potassium iodide inhibits neutrophil chemotaxis. · 1990 · https://pubmed.ncbi.nlm.nih.gov/1972841/
    Complete structured claim and evidence

  2. Following the four-week kiwifruit intervention, stimulated neutrophil chemotaxis increased about 20% in paired ex vivo assays (n=12, p=0.041); unstimulated migration did not change.

    Dietary SunGold kiwifruit intervention → Neutrophil chemotaxis source_derived_draftungraded
    Experimental context and source evidence
    cross_nutrient
    false
    experimental_model
    Young men with suboptimal plasma vitamin C; before/after kiwifruit intervention and isolated neutrophils
    exposure
    Two SunGold kiwifruit/day for four weeks in men with initially suboptimal plasma vitamin C
    limitations
    Uncontrolled whole-food intervention; assay improvement does not establish fewer infections or a vitamin C-specific causal mechanism.
    nutrient_topic
    Vitamin C research collection; topical membership is not evidence of a direct dietary effect. · Vitamin C
    organism
    Homo sapiens
    plain_language
    Participants’ isolated neutrophils moved more effectively toward the laboratory stimulus after the intervention.
    primary_references
    [bozonet2015] Enhanced human neutrophil vitamin C status, chemotaxis and oxidant generation following dietary supplementation with vitamin C-rich SunGold kiwifruit. (2015). https://pubmed.ncbi.nlm.nih.gov/25912037/ DOI: 10.3390/nu7042574
    tissue_or_cell_type
    Isolated peripheral-blood neutrophils

    Vitamin C: mechanisms, deficiency and nutrient interactions (2026-09-17) · lines 559–570

    AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · Young men with suboptimal plasma vitamin C; before/after kiwifruit intervention and isolated neutrophils · source_derived_draft · unverified_draft

    ### vc-transport-kiwifruit-neutrophil-chemotaxis Following the four-week kiwifruit intervention, stimulated neutrophil chemotaxis increased about 20% in paired ex vivo assays (n=12, p=0.041); unstimulated migration did not change. Condition category: normal nutrient_topic: Vitamin C research collection; topical membership is not evidence of a direct dietary effect. plain_language: Participants’ isolated neutrophils moved more effectively toward the laboratory stimulus after the intervention. organism: Homo sapiens tissue_or_cell_type: Isolated peripheral-blood neutrophils experimental_model: Young men with suboptimal plasma vitamin C; before/after kiwifruit intervention and isolated neutrophils limitations: Uncontrolled whole-food intervention; assay improvement does not establish fewer infections or a vitamin C-specific causal mechanism. exposure: Two SunGold kiwifruit/day for four weeks in men with initially suboptimal plasma vitamin C cross_nutrient: false [bozonet2015] Enhanced human neutrophil vitamin C status, chemotaxis and oxidant generation following dietary supplementation with vitamin C-rich SunGold kiwifruit. (2015). https://pubmed.ncbi.nlm.nih.gov/25912037/ DOI: 10.3390/nu7042574
    Complete structured claim and evidence

In the sources

Preserved passages that mention this component, quoted exactly. Open one to read it in context.

    This is a research prototype built from draft material. It is not medical advice, and its statements still await verification against the original studies.

    Evidence, AI assistance and curation standards