Component
Human subjective satiety
Human subjective satiety. Species, exposure and limitations are retained in each linked claim.
2 recorded relationships. Experimental role, claim status and evidence remain attached to each record.
How nutrients influence it
Every nutrient with a recorded effect on this component, credited to the nutrient that acted rather than the chapter that recorded it. Open a nutrient to see the findings and the conditions they were measured under.
Other things that act on it
Enzymes, hormones, genes, and other components with a recorded effect. These are not nutrients, so they do not count toward the arrows above. Each finding names the chapter that recorded it.
How nutrients reach it in more than one step
Chains of two or more recorded steps that end here, grouped by the nutrient they start from. Each step is a separate finding, so a chain is a route a mechanism could take, not proof that it does.
Tracing routes…
What it does
Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.
What acts on it
Pectin was associated with increased reported satiety in this experiment.
Experimental context and source evidence
- evidence_span
- {"source_cache": "artifacts/pectin-research/2689931.abstract.txt", "locator": "Primary indexed abstract; zero-based, end-exclusive Unicode character offsets", "file_sha256": "5f3b008530bce6948a60fde1f68eff60e2bbec7ba0cc46b4bce2e56cd8313513", "start_char": 0, "end_char": 277, "text_sha256": "5f3b008530bce6948a60fde1f68eff60e2bbec7ba0cc46b4bce2e56cd8313513"}
- experimental_model
- Meal study with scintigraphic gastric emptying
- exposure
- Pectin compared with methylcellulose; dose not resolved in indexed abstract
- limitations
- Small context-specific study; subjective satiety does not establish durable weight loss.
- nutrient_topic
- Pectin research collection; topical membership is not evidence of a direct dietary effect. · Pectin, structurally heterogeneous plant polysaccharides
- organism
- Homo sapiens
- plain_language
- Participants reported feeling fuller.
- primary_references
- [pectin-p2689931] Pectin delays gastric emptying. (1989). https://pubmed.ncbi.nlm.nih.gov/2689931/ DOI: 10.1111/j.1753-4887.1989.tb02860.x
- tissue_or_cell_type
- Stomach, satiety and plasma hormones in obesity
Pectin: metabolism, signaling and nutrient connections (2026-09-17) · lines 828–839
AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · Meal study with scintigraphic gastric emptying · source_derived_draft · unverified_draft
### pectin-satiety Pectin was associated with increased reported satiety in this experiment. Condition category: normal nutrient_topic: Pectin research collection; topical membership is not evidence of a direct dietary effect. plain_language: Participants reported feeling fuller. organism: Homo sapiens tissue_or_cell_type: Stomach, satiety and plasma hormones in obesity experimental_model: Meal study with scintigraphic gastric emptying limitations: Small context-specific study; subjective satiety does not establish durable weight loss. exposure: Pectin compared with methylcellulose; dose not resolved in indexed abstract evidence_span: {"source_cache": "artifacts/pectin-research/2689931.abstract.txt", "locator": "Primary indexed abstract; zero-based, end-exclusive Unicode character offsets", "file_sha256": "5f3b008530bce6948a60fde1f68eff60e2bbec7ba0cc46b4bce2e56cd8313513", "start_char": 0, "end_char": 277, "text_sha256": "5f3b008530bce6948a60fde1f68eff60e2bbec7ba0cc46b4bce2e56cd8313513"} [pectin-p2689931] Pectin delays gastric emptying. (1989). https://pubmed.ncbi.nlm.nih.gov/2689931/ DOI: 10.1111/j.1753-4887.1989.tb02860.x
Complete structured claim and evidence
Where it participates (unsigned role)
Oat beta-glucan increased feelings of fullness and satiety but did not affect energy and amount eaten at the ad libitum test meal, and there was a treatment by time interaction for plasma GLP-1, plasma insulin and blood glucose, with GLP-1 significantly reduced at 90 minutes, blood glucose at 30 minutes and plasma insulin at 30 and 60 minutes following the oat beta-glucan breakfast compared with the control breakfast, so four grams of high molecular weight oat beta-glucan lowers appetite but not ad libitum eating and beneficially modulates postprandial glycaemia, it does however not increase plasma GLP-1 secretion.
Experimental context and source evidence
- evidence_span
- {"source_cache": "artifacts/glucan-research/29920323.abstract.txt", "locator": "Indexed abstract; zero-based, end-exclusive Unicode character offsets", "file_sha256": "8038ec81d42b26e4b4892cd24cd0ce26597ba28ef57296a90062428f6ecbceb4", "start_char": 0, "end_char": 1413, "text_sha256": "8038ec81d42b26e4b4892cd24cd0ce26597ba28ef57296a90062428f6ecbceb4"}
- experimental_model
- Randomised double-blind crossover trial in 33 normal-weight subjects with an unrestricted test meal
- exposure
- A breakfast containing 4 grams of high molecular weight oat beta-glucan against a control breakfast
- limitations
- An acute crossover in normal-weight subjects. Fullness and satiety rose without any change in what was actually eaten afterwards.
- nutrient_topic
- Beta-glucan research collection; topical membership is not evidence of a direct clinical effect, and each preparation is recorded as its own entity with no family link joining any pair. · Beta-glucan
- organism
- Human
- plain_language
- People felt fuller and ate the same amount, and the gut hormone usually credited for fullness went down, not up.
- primary_references
- [bg-p29920323] Effects of oat β-glucan consumption at breakfast on ad libitum eating, appetite, glycemia, insulinemia and GLP-1 concentrations in healthy subjects. (2018). https://pubmed.ncbi.nlm.nih.gov/29920323/ DOI: 10.1016/j.appet.2018.06.019
- tissue_or_cell_type
- Postprandial circulation and appetite
AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · Randomised double-blind crossover trial in 33 normal-weight subjects with an unrestricted test meal · source_derived_draft · unverified_draft
### bg-satiety-rises-but-glp1-falls Oat beta-glucan increased feelings of fullness and satiety but did not affect energy and amount eaten at the ad libitum test meal, and there was a treatment by time interaction for plasma GLP-1, plasma insulin and blood glucose, with GLP-1 significantly reduced at 90 minutes, blood glucose at 30 minutes and plasma insulin at 30 and 60 minutes following the oat beta-glucan breakfast compared with the control breakfast, so four grams of high molecular weight oat beta-glucan lowers appetite but not ad libitum eating and beneficially modulates postprandial glycaemia, it does however not increase plasma GLP-1 secretion. Condition category: normal nutrient_topic: Beta-glucan research collection; topical membership is not evidence of a direct clinical effect, and each preparation is recorded as its own entity with no family link joining any pair. plain_language: People felt fuller and ate the same amount, and the gut hormone usually credited for fullness went down, not up. organism: Human tissue_or_cell_type: Postprandial circulation and appetite experimental_model: Randomised double-blind crossover trial in 33 normal-weight subjects with an unrestricted test meal limitations: An acute crossover in normal-weight subjects. Fullness and satiety rose without any change in what was actually eaten afterwards. exposure: A breakfast containing 4 grams of high molecular weight oat beta-glucan against a control breakfast evidence_span: {"source_cache": "artifacts/glucan-research/29920323.abstract.txt", "locator": "Indexed abstract; zero-based, end-exclusive Unicode character offsets", "file_sha256": "8038ec81d42b26e4b4892cd24cd0ce26597ba28ef57296a90062428f6ecbceb4", "start_char": 0, "end_char": 1413, "text_sha256": "8038ec81d42b26e4b4892cd24cd0ce26597ba28ef57296a90062428f6ecbceb4"} [bg-p29920323] Effects of oat β-glucan consumption at breakfast on ad libitum eating, appetite, glycemia, insulinemia and GLP-1 concentrations in healthy subjects. (2018). https://pubmed.ncbi.nlm.nih.gov/29920323/ DOI: 10.1016/j.appet.2018.06.019
Complete structured claim and evidence
The events it takes part in
A mechanism often involves more than two components. These are the full events, with every participant and its role.
Situations it appears in
Low-supply and faulty-machinery situations recorded in the chapters where this component plays a part.
In the sources
Preserved passages that mention this component, quoted exactly. Open one to read it in context.
Open hypotheses
Proposed ideas that involve this component. They are labeled as hypotheses and do not change any recorded statement.
This is a research prototype built from draft material. It is not medical advice, and its statements still await verification against the original studies.