Component

Plasma aldosterone concentration

Independent biological entity. Read linked claims for experimental scope and context.

1 recorded relationships. Experimental role, claim status and evidence remain attached to each record.

How nutrients influence it

Every nutrient with a recorded effect on this component, credited to the nutrient that acted rather than the chapter that recorded it. Open a nutrient to see the findings and the conditions they were measured under.

How nutrients reach it in more than one step

Chains of two or more recorded steps that end here, grouped by the nutrient they start from. Each step is a separate finding, so a chain is a route a mechanism could take, not proof that it does.

Tracing routes…

What it does

Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.

Recorded relationships

What acts on it

  1. Potassium increased aldosterone, renin and copeptin in the post-hoc hormone analysis while blood pressure fell.

    Potassium → Plasma aldosterone concentration source_derived_draftungraded
    Experimental context and source evidence
    cross_nutrient
    Potassium -> endocrine regulation; sodium/volume context retained.
    experimental_model
    35 participants from the same feeding trial.
    limitations
    Post-hoc circulating markers do not identify receptor signaling or a universal endocrine response.
    nutrient_topic
    Potassium research collection; topical membership is not evidence of a direct dietary effect. · Potassium
    organism
    Homo sapiens
    plain_language
    A hormone response can accompany a pressure reduction rather than imply the opposite outcome.
    primary_references
    [k-gijsbers2016-hormones] Effects of potassium supplementation on markers of osmoregulation and volume regulation: results of a fully controlled dietary intervention study (2016). https://pubmed.ncbi.nlm.nih.gov/26599222/ DOI: 10.1097/hjh.0000000000000786
    tissue_or_cell_type
    Plasma

    Potassium: cross-nutrient mechanisms and deficiency (2026-09-17) · lines 1568–1578

    AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · 35 participants from the same feeding trial. · source_derived_draft · unverified_draft

    ### k-controlled-feeding-regulatory-hormones Potassium increased aldosterone, renin and copeptin in the post-hoc hormone analysis while blood pressure fell. Condition category: normal nutrient_topic: Potassium research collection; topical membership is not evidence of a direct dietary effect. plain_language: A hormone response can accompany a pressure reduction rather than imply the opposite outcome. organism: Homo sapiens tissue_or_cell_type: Plasma experimental_model: 35 participants from the same feeding trial. limitations: Post-hoc circulating markers do not identify receptor signaling or a universal endocrine response. cross_nutrient: Potassium -> endocrine regulation; sodium/volume context retained. [k-gijsbers2016-hormones] Effects of potassium supplementation on markers of osmoregulation and volume regulation: results of a fully controlled dietary intervention study (2016). https://pubmed.ncbi.nlm.nih.gov/26599222/ DOI: 10.1097/hjh.0000000000000786
    Complete structured claim and evidence

In the sources

Preserved passages that mention this component, quoted exactly. Open one to read it in context.

    This is a research prototype built from draft material. It is not medical advice, and its statements still await verification against the original studies.

    Evidence, AI assistance and curation standards