Component

Calculated whole-body iron stores

Independent biological entity. Read linked claims for experimental scope and context.

1 recorded relationships. Experimental role, claim status and evidence remain attached to each record.

How nutrients influence it

Every nutrient with a recorded effect on this component, credited to the nutrient that acted rather than the chapter that recorded it. Open a nutrient to see the findings and the conditions they were measured under.

How nutrients reach it in more than one step

Chains of two or more recorded steps that end here, grouped by the nutrient they start from. Each step is a separate finding, so a chain is a route a mechanism could take, not proof that it does.

Tracing routes…

What it does

Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.

Recorded relationships

Where it participates (unsigned role)

  1. Ferritin fell while calculated total-body iron remained unchanged in the same trial, consistent with redistribution of existing iron.

    Vitamin A → Serum ferritin concentration source_derived_draftungraded
    Experimental context and source evidence
    availability_state
    nutrient_deficiency Imported condition classification; unverified.
    experimental_model
    Ferritin/transferrin-receptor-based estimates.
    limitations
    Redistribution is an interpretation; liver iron was not directly biopsied.
    nutrient_topic
    Vitamin A research collection; topical membership is not evidence of a direct dietary effect. · Vitamin A
    organism
    Homo sapiens
    plain_language
    A better blood count did not require a measured increase in total-body iron.
    primary_references
    [va-zimmermann2006-iron] Vitamin A supplementation in children with poor vitamin A and iron status increases erythropoietin and hemoglobin concentrations without changing total body iron (2006). https://pubmed.ncbi.nlm.nih.gov/16960172/ DOI: 10.1093/ajcn/84.3.580
    tissue_or_cell_type
    Blood; inferred storage
    trigger_kind
    nutrient_deficiency Imported condition classification; unverified.

    Vitamin A: forms, mechanisms, deficiency and excess (2026-09-17) · lines 1472–1481

    AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · Ferritin/transferrin-receptor-based estimates. · source_derived_draft · unverified_draft

    ### va-repletion-iron-store-redistribution Ferritin fell while calculated total-body iron remained unchanged in the same trial, consistent with redistribution of existing iron. Condition category: nutrient_deficiency nutrient_topic: Vitamin A research collection; topical membership is not evidence of a direct dietary effect. plain_language: A better blood count did not require a measured increase in total-body iron. organism: Homo sapiens tissue_or_cell_type: Blood; inferred storage experimental_model: Ferritin/transferrin-receptor-based estimates. limitations: Redistribution is an interpretation; liver iron was not directly biopsied. [va-zimmermann2006-iron] Vitamin A supplementation in children with poor vitamin A and iron status increases erythropoietin and hemoglobin concentrations without changing total body iron (2006). https://pubmed.ncbi.nlm.nih.gov/16960172/ DOI: 10.1093/ajcn/84.3.580
    Complete structured claim and evidence

In the sources

Preserved passages that mention this component, quoted exactly. Open one to read it in context.

    This is a research prototype built from draft material. It is not medical advice, and its statements still await verification against the original studies.

    Evidence, AI assistance and curation standards