Component
Autoimmune thyroiditis incidence
Autoimmune thyroiditis incidence. Study-specific scope and measurement methods are recorded with each claim.
1 recorded relationships. Experimental role, claim status and evidence remain attached to each record.
How nutrients influence it
Every nutrient with a recorded effect on this component, credited to the nutrient that acted rather than the chapter that recorded it. Open a nutrient to see the findings and the conditions they were measured under.
Other things that act on it
Enzymes, hormones, genes, and other components with a recorded effect. These are not nutrients, so they do not count toward the arrows above. Each finding names the chapter that recorded it.
How nutrients reach it in more than one step
Chains of two or more recorded steps that end here, grouped by the nutrient they start from. Each step is a separate finding, so a chain is a route a mechanism could take, not proof that it does.
Tracing routes…
What it does
Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.
What acts on it
Five-year autoimmune thyroiditis incidence was 0.2%, 1.0% and 1.3% across the three increasing regional iodine-exposure categories.
Experimental context and source evidence
- cross_nutrient
- false
- evidence_location
- Primary indexed abstract.
- experimental_model
- Prospective five-year observational follow-up in three Chinese regions;3761 enrolled and 3018 followed
- exposure
- Regional baseline median urinary iodine 84, 243 and 651 micrograms/L; followed 1999–2004.
- limitations
- Regional exposure is not randomized individual dosing; confounding and baseline susceptibility may contribute. Median urinary iodine defines cohort exposure, not an individual disease threshold. Association does not establish the immune mechanism or show that every person with antibodies responds identically to iodine.
- nutrient_topic
- Iodine research collection; topical membership is not evidence of a direct dietary effect. · Iodine
- organism
- Homo sapiens
- plain_language
- The same cohort also found more autoimmune thyroiditis in the higher-exposure regions.
- primary_references
- [iod-clin-teng2006] Effect of iodine intake on thyroid diseases in China. (2006). https://pubmed.ncbi.nlm.nih.gov/16807415/ DOI: 10.1056/nejmoa054022
- tissue_or_cell_type
- Serum thyroid function/antibodies and thyroid ultrasound
Iodine: thyroid hormone production, deficiency, excess and nutrient interactions (2026-09-17) · lines 1496–1508
AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · Prospective five-year observational follow-up in three Chinese regions;3761 enrolled and 3018 followed · source_derived_draft · unverified_draft
### iod-clin-regional-autoimmunity Five-year autoimmune thyroiditis incidence was 0.2%, 1.0% and 1.3% across the three increasing regional iodine-exposure categories. Condition category: normal nutrient_topic: Iodine research collection; topical membership is not evidence of a direct dietary effect. plain_language: The same cohort also found more autoimmune thyroiditis in the higher-exposure regions. organism: Homo sapiens tissue_or_cell_type: Serum thyroid function/antibodies and thyroid ultrasound experimental_model: Prospective five-year observational follow-up in three Chinese regions;3761 enrolled and 3018 followed limitations: Regional exposure is not randomized individual dosing; confounding and baseline susceptibility may contribute. Median urinary iodine defines cohort exposure, not an individual disease threshold. Association does not establish the immune mechanism or show that every person with antibodies responds identically to iodine. exposure: Regional baseline median urinary iodine 84, 243 and 651 micrograms/L; followed 1999–2004. cross_nutrient: false evidence_location: Primary indexed abstract. [iod-clin-teng2006] Effect of iodine intake on thyroid diseases in China. (2006). https://pubmed.ncbi.nlm.nih.gov/16807415/ DOI: 10.1056/nejmoa054022
Complete structured claim and evidence
The events it takes part in
A mechanism often involves more than two components. These are the full events, with every participant and its role.
Situations it appears in
Low-supply and faulty-machinery situations recorded in the chapters where this component plays a part.
In the sources
Preserved passages that mention this component, quoted exactly. Open one to read it in context.
Open hypotheses
Proposed ideas that involve this component. They are labeled as hypotheses and do not change any recorded statement.
This is a research prototype built from draft material. It is not medical advice, and its statements still await verification against the original studies.