Component
Urinary riboflavin excretion
Urinary riboflavin excretion. Species, exposure and limitations are retained in each linked claim.
1 recorded relationships. Experimental role, claim status and evidence remain attached to each record.
How nutrients influence it
Every nutrient with a recorded effect on this component, credited to the nutrient that acted rather than the chapter that recorded it. Open a nutrient to see the findings and the conditions they were measured under.
Other things that act on it
Enzymes, hormones, genes, and other components with a recorded effect. These are not nutrients, so they do not count toward the arrows above. Each finding names the chapter that recorded it.
How nutrients reach it in more than one step
Chains of two or more recorded steps that end here, grouped by the nutrient they start from. Each step is a separate finding, so a chain is a route a mechanism could take, not proof that it does.
Tracing routes…
What it does
Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.
What acts on it
Approximately two thirds of 14 patients evaluated after boric-acid ingestion had markedly increased urinary riboflavin excretion.
Experimental context and source evidence
- evidence_span
- {"source_cache": "artifacts/boron-research/659962.abstract.txt", "locator": "Exact primary indexed abstract; zero-based, end-exclusive Unicode character offsets", "file_sha256": "903bb51f45f538c7ce8d0a73e5247e4619efb74ce4cd3fa8fc06f9bf108ba92f", "start_char": 0, "end_char": 696, "text_sha256": "903bb51f45f538c7ce8d0a73e5247e4619efb74ce4cd3fa8fc06f9bf108ba92f"}
- experimental_model
- Poison-control case series; 14 children and adults
- exposure
- Poisoning-related ingestion; most increased urinary riboflavin appeared in the first 24 hours
- limitations
- Uncontrolled case series, not ordinary dietary exposure. Increased excretion does not alone establish tissue deficiency or the responsible molecular transport mechanism.
- nutrient_topic
- Boron research collection; topical membership is not evidence of a direct dietary effect. · Boron
- organism
- Human
- plain_language
- High boric-acid exposure was associated with loss of vitamin B2 into urine in this case series.
- primary_references
- [boron-p659962] Increased urinary riboflavin excretion resulting from boric acid ingestion. (1978). https://pubmed.ncbi.nlm.nih.gov/659962/
- tissue_or_cell_type
- Urine after boric-acid ingestion
Boron: chemistry, nutrient interactions, low-intake studies and mechanistic uncertainties (2026-09-17) · lines 937–948
AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · Poison-control case series; 14 children and adults · source_derived_draft · unverified_draft
### boron-poisoning-riboflavinuria Approximately two thirds of 14 patients evaluated after boric-acid ingestion had markedly increased urinary riboflavin excretion. Condition category: normal nutrient_topic: Boron research collection; topical membership is not evidence of a direct dietary effect. plain_language: High boric-acid exposure was associated with loss of vitamin B2 into urine in this case series. organism: Human tissue_or_cell_type: Urine after boric-acid ingestion experimental_model: Poison-control case series; 14 children and adults limitations: Uncontrolled case series, not ordinary dietary exposure. Increased excretion does not alone establish tissue deficiency or the responsible molecular transport mechanism. exposure: Poisoning-related ingestion; most increased urinary riboflavin appeared in the first 24 hours evidence_span: {"source_cache": "artifacts/boron-research/659962.abstract.txt", "locator": "Exact primary indexed abstract; zero-based, end-exclusive Unicode character offsets", "file_sha256": "903bb51f45f538c7ce8d0a73e5247e4619efb74ce4cd3fa8fc06f9bf108ba92f", "start_char": 0, "end_char": 696, "text_sha256": "903bb51f45f538c7ce8d0a73e5247e4619efb74ce4cd3fa8fc06f9bf108ba92f"} [boron-p659962] Increased urinary riboflavin excretion resulting from boric acid ingestion. (1978). https://pubmed.ncbi.nlm.nih.gov/659962/
Complete structured claim and evidence
The events it takes part in
A mechanism often involves more than two components. These are the full events, with every participant and its role.
Situations it appears in
Low-supply and faulty-machinery situations recorded in the chapters where this component plays a part.
In the sources
Preserved passages that mention this component, quoted exactly. Open one to read it in context.
Open hypotheses
Proposed ideas that involve this component. They are labeled as hypotheses and do not change any recorded statement.
This is a research prototype built from draft material. It is not medical advice, and its statements still await verification against the original studies.