Component

Plasma H2S readout in the SAC infarction rat study

Context-specific entity; species, compartment and exposure are stated on each claim.

1 recorded relationships. Experimental role, claim status and evidence remain attached to each record.

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What it does

Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.

Recorded relationships

What acts on it

  1. SAC-treated infarcted rats had higher measured plasma H2S than controls and SAC-plus-PAG rats.

    Experimental context and source evidence
    acting_entity
    s-allylcysteine
    dose
    SAC 50 mg/kg/day; PAG 10 mg/kg/day
    duration
    Seven-day pretreatment; assessed 48 hours after infarction
    evidence_access
    Primary abstract
    experimental_comparison
    Saline, SAC, SAC plus PAG, and PAG-alone groups
    experimental_model
    Acute myocardial infarction; left ventricular and plasma measurements
    interpretation_status
    Source-derived research curation; not independent primary verification
    limitations
    Historical sulfide assay result; exact chemical species and analytical selectivity require full-text assessment.
    nutrient_topic
    S-allylcysteine collection; species, compartment, exposure, co-substrates and manipulation remain explicit. · S-allyl-L-cysteine / SAC
    organism
    Rattus norvegicus
    plain_language
    The sulfur signal had its own measured endpoint.
    primary_references
    [17766469] S-allylcysteine mediates cardioprotection in an acute myocardial infarction rat model via a hydrogen sulfide-mediated pathway. · 2007 · https://pubmed.ncbi.nlm.nih.gov/17766469/ · DOI 10.1152/ajpheart.00853.2007
    route
    Administration route not specified in accessed abstract
    tissue_or_cell_type
    Acute myocardial infarction; left ventricular and plasma measurements

    S-allylcysteine: sulfur signaling, redox responses and cross-nutrient mechanisms (2026-09-20) · lines 248–255

    AI-assisted research curation; primary references, access levels and experimental limitations individually identified. Not publisher full text. · supports · Acute myocardial infarction; left ventricular and plasma measurements · source_derived_draft · unverified_draft

    ## s-allylcysteine-rat-h2s The sulfur signal had its own measured endpoint. SAC-treated infarcted rats had higher measured plasma H2S than controls and SAC-plus-PAG rats. Model: Acute myocardial infarction; left ventricular and plasma measurements Limitations: Historical sulfide assay result; exact chemical species and analytical selectivity require full-text assessment. Evidence access: Primary abstract [17766469] S-allylcysteine mediates cardioprotection in an acute myocardial infarction rat model via a hydrogen sulfide-mediated pathway. · 2007 · https://pubmed.ncbi.nlm.nih.gov/17766469/ · DOI 10.1152/ajpheart.00853.2007 Structured context: {"organism": "Rattus norvegicus", "tissue_or_cell_type": "Acute myocardial infarction; left ventricular and plasma measurements", "dose": "SAC 50 mg/kg/day; PAG 10 mg/kg/day", "duration": "Seven-day pretreatment; assessed 48 hours after infarction", "route": "Administration route not specified in accessed abstract", "experimental_comparison": "Saline, SAC, SAC plus PAG, and PAG-alone groups", "acting_entity": "s-allylcysteine", "interpretation_status": "Source-derived research curation; not independent primary verification"}
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In the sources

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