Component

Methyl linoleate

Methyl ester of linoleic acid used as an oxidizable chemical substrate.

1 recorded relationships. Experimental role, claim status and evidence remain attached to each record.

How nutrients influence it

Every nutrient with a recorded effect on this component, credited to the nutrient that acted rather than the chapter that recorded it. Open a nutrient to see the findings and the conditions they were measured under.

How nutrients reach it in more than one step

Chains of two or more recorded steps that end here, grouped by the nutrient they start from. Each step is a separate finding, so a chain is a route a mechanism could take, not proof that it does.

Tracing routes…

What it does

Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.

Recorded relationships

Where it participates (unsigned role)

  1. In azo-initiated methyl linoleate oxidation in solution, alpha-tocopherol interrupted the radical chain; the reported inhibition rate constant was 5.1 × 10^5 M^-1 s^-1 at 37 °C, with kinetics consistent with trapping two peroxyl radicals per molecule under those conditions.

    Alpha-tocopherol → Lipid peroxyl radicals source_derived_draftungraded
    Experimental context and source evidence
    cross_nutrient
    false
    evidence_location
    Primary abstract
    experimental_model
    Solution autoxidation kinetics
    exposure
    Azo initiators; 37 °C.
    limitations
    Solution-specific kinetics and inferred stoichiometry, not a universal membrane rate.
    nutrient_topic
    Vitamin E research collection; topical membership is not evidence of a direct dietary effect. · Vitamin E
    organism
    Cell-free
    plain_language
    Vitamin E intercepted radicals that would otherwise continue oxidizing this fatty-acid ester.
    primary_references
    [ver-niki1984] Inhibition of oxidation of methyl linoleate in solution by vitamin E and vitamin C. (1984). https://pubmed.ncbi.nlm.nih.gov/6706998/ DOI: 10.1016/s0021-9258(17)43026-2
    tissue_or_cell_type
    Organic solution

    Vitamin E: transport, membrane protection and nutrient interactions (2026-09-17) · lines 480–492

    AI-assisted literature curation; primary study URLs and scope retained in the document and extraction. Not publisher full text. · supports · Solution autoxidation kinetics · source_derived_draft · unverified_draft

    ### ver-peroxyl-trapping In azo-initiated methyl linoleate oxidation in solution, alpha-tocopherol interrupted the radical chain; the reported inhibition rate constant was 5.1 × 10^5 M^-1 s^-1 at 37 °C, with kinetics consistent with trapping two peroxyl radicals per molecule under those conditions. Condition category: normal nutrient_topic: Vitamin E research collection; topical membership is not evidence of a direct dietary effect. plain_language: Vitamin E intercepted radicals that would otherwise continue oxidizing this fatty-acid ester. organism: Cell-free tissue_or_cell_type: Organic solution experimental_model: Solution autoxidation kinetics limitations: Solution-specific kinetics and inferred stoichiometry, not a universal membrane rate. exposure: Azo initiators; 37 °C. cross_nutrient: false evidence_location: Primary abstract [ver-niki1984] Inhibition of oxidation of methyl linoleate in solution by vitamin E and vitamin C. (1984). https://pubmed.ncbi.nlm.nih.gov/6706998/ DOI: 10.1016/s0021-9258(17)43026-2
    Complete structured claim and evidence

In the sources

Preserved passages that mention this component, quoted exactly. Open one to read it in context.

    This is a research prototype built from draft material. It is not medical advice, and its statements still await verification against the original studies.

    Evidence, AI assistance and curation standards