Component
Myricitrin-metabolizing intestinal isolates from one human donor
Context-specific entity; species, compartment and exposure are stated on each claim.
3 recorded relationships. Experimental role, claim status and evidence remain attached to each record.
How nutrients influence it
Every nutrient with a recorded effect on this component, credited to the nutrient that acted rather than the chapter that recorded it. Open a nutrient to see the findings and the conditions they were measured under.
Other things that act on it
Enzymes, hormones, genes, and other components with a recorded effect. These are not nutrients, so they do not count toward the arrows above. Each finding names the chapter that recorded it.
How nutrients reach it in more than one step
Chains of two or more recorded steps that end here, grouped by the nutrient they start from. Each step is a separate finding, so a chain is a route a mechanism could take, not proof that it does.
Tracing routes…
What it does
Every recorded relationship this component is part of, grouped by its role. Plain wording comes first; the technical statement follows.
Where it participates (unsigned role)
Human-donor intestinal bacterial cultures yielded Myricetin through the reported deglycosylation route.
Experimental context and source evidence
- evidence_access
- Primary abstract
- experimental_model
- Four active isolates selected from about 100 colonies from one healthy female donor; most identified as Escherichia.
- limitations
- Mass-spectrometric pathway assignment; no specific enzyme or population-wide conversion rate established.
- nutrient_topic
- Myricetin collection; molecular form, preparation, species, exposure and manipulation remain explicit. · Myricetin
- plain_language
- Gut processing produces a chemically different molecule.
- primary_references
- Identification of the metabolites of myricitrin produced by human intestinal bacteria in vitro using ultra-performance liquid chromatography/quadrupole time-of-flight mass spectrometry. · 2014 · https://pubmed.ncbi.nlm.nih.gov/24882500/ · DOI 10.1517/17425255.2014.918954
Myricetin: metabolism, immune signaling, redox chemistry and cross-nutrient mechanisms (2026-09-19) · lines 20–26
AI-assisted research curation; primary references, access levels and experimental limitations individually identified. Not publisher full text. · supports · Four active isolates selected from about 100 colonies from one healthy female donor; most identified as Escherichia. · source_derived_draft · unverified_draft
## myricetin-microbial-deglycosylation Gut processing produces a chemically different molecule. Human-donor intestinal bacterial cultures yielded Myricetin through the reported deglycosylation route. Model: Four active isolates selected from about 100 colonies from one healthy female donor; most identified as Escherichia. Limitations: Mass-spectrometric pathway assignment; no specific enzyme or population-wide conversion rate established. Evidence access: Primary abstract Identification of the metabolites of myricitrin produced by human intestinal bacteria in vitro using ultra-performance liquid chromatography/quadrupole time-of-flight mass spectrometry. · 2014 · https://pubmed.ncbi.nlm.nih.gov/24882500/ · DOI 10.1517/17425255.2014.918954
Complete structured claim and evidenceHuman-donor intestinal bacterial cultures yielded Quercitrin / quercetin 3-O-rhamnoside through the reported dehydroxylation route.
Experimental context and source evidence
- evidence_access
- Primary abstract
- experimental_model
- Four active isolates selected from about 100 colonies from one healthy female donor; most identified as Escherichia.
- limitations
- Mass-spectrometric pathway assignment; no specific enzyme or population-wide conversion rate established.
- nutrient_topic
- Myricetin collection; molecular form, preparation, species, exposure and manipulation remain explicit. · Myricetin
- plain_language
- Gut processing produces a chemically different molecule.
- primary_references
- Identification of the metabolites of myricitrin produced by human intestinal bacteria in vitro using ultra-performance liquid chromatography/quadrupole time-of-flight mass spectrometry. · 2014 · https://pubmed.ncbi.nlm.nih.gov/24882500/ · DOI 10.1517/17425255.2014.918954
Myricetin: metabolism, immune signaling, redox chemistry and cross-nutrient mechanisms (2026-09-19) · lines 28–34
AI-assisted research curation; primary references, access levels and experimental limitations individually identified. Not publisher full text. · supports · Four active isolates selected from about 100 colonies from one healthy female donor; most identified as Escherichia. · source_derived_draft · unverified_draft
## myricetin-microbial-dehydroxylation Gut processing produces a chemically different molecule. Human-donor intestinal bacterial cultures yielded Quercitrin / quercetin 3-O-rhamnoside through the reported dehydroxylation route. Model: Four active isolates selected from about 100 colonies from one healthy female donor; most identified as Escherichia. Limitations: Mass-spectrometric pathway assignment; no specific enzyme or population-wide conversion rate established. Evidence access: Primary abstract Identification of the metabolites of myricitrin produced by human intestinal bacteria in vitro using ultra-performance liquid chromatography/quadrupole time-of-flight mass spectrometry. · 2014 · https://pubmed.ncbi.nlm.nih.gov/24882500/ · DOI 10.1517/17425255.2014.918954
Complete structured claim and evidenceHuman-donor intestinal bacterial cultures yielded Quercetin through the reported subsequent deglycosylation route.
Experimental context and source evidence
- evidence_access
- Primary abstract
- experimental_model
- Four active isolates selected from about 100 colonies from one healthy female donor; most identified as Escherichia.
- limitations
- Mass-spectrometric pathway assignment; no specific enzyme or population-wide conversion rate established.
- nutrient_topic
- Myricetin collection; molecular form, preparation, species, exposure and manipulation remain explicit. · Myricetin
- plain_language
- Gut processing produces a chemically different molecule.
- primary_references
- Identification of the metabolites of myricitrin produced by human intestinal bacteria in vitro using ultra-performance liquid chromatography/quadrupole time-of-flight mass spectrometry. · 2014 · https://pubmed.ncbi.nlm.nih.gov/24882500/ · DOI 10.1517/17425255.2014.918954
Myricetin: metabolism, immune signaling, redox chemistry and cross-nutrient mechanisms (2026-09-19) · lines 36–42
AI-assisted research curation; primary references, access levels and experimental limitations individually identified. Not publisher full text. · supports · Four active isolates selected from about 100 colonies from one healthy female donor; most identified as Escherichia. · source_derived_draft · unverified_draft
## myricetin-microbial-quercetin Gut processing produces a chemically different molecule. Human-donor intestinal bacterial cultures yielded Quercetin through the reported subsequent deglycosylation route. Model: Four active isolates selected from about 100 colonies from one healthy female donor; most identified as Escherichia. Limitations: Mass-spectrometric pathway assignment; no specific enzyme or population-wide conversion rate established. Evidence access: Primary abstract Identification of the metabolites of myricitrin produced by human intestinal bacteria in vitro using ultra-performance liquid chromatography/quadrupole time-of-flight mass spectrometry. · 2014 · https://pubmed.ncbi.nlm.nih.gov/24882500/ · DOI 10.1517/17425255.2014.918954
Complete structured claim and evidence
The events it takes part in
A mechanism often involves more than two components. These are the full events, with every participant and its role.
Situations it appears in
Low-supply and faulty-machinery situations recorded in the chapters where this component plays a part.
In the sources
Preserved passages that mention this component, quoted exactly. Open one to read it in context.
Open hypotheses
Proposed ideas that involve this component. They are labeled as hypotheses and do not change any recorded statement.
This is a research prototype built from draft material. It is not medical advice, and its statements still await verification against the original studies.