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High salicylate concentrations, which the authors relate to high-dose therapy rather than to antiplatelet dosing.\nexposure: Sodium salicylate against tumour necrosis factor alpha stimulation, with indomethacin as a cyclooxygenase control\nevidence_span: {\"source_cache\": \"artifacts/aspirin-research/8621937.abstract.txt\", \"locator\": \"Indexed abstract; zero-based, end-exclusive Unicode character offsets\", \"file_sha256\": \"9b5aff676845bd0a47f25a0f63564880f70af7667b5ab9d667cece58d05356f1\", \"start_char\": 0, \"end_char\": 1767, \"text_sha256\": \"9b5aff676845bd0a47f25a0f63564880f70af7667b5ab9d667cece58d05356f1\"}\n[asa-p8621937] Salicylates inhibit I kappa B-alpha phosphorylation, endothelial-leukocyte adhesion molecule expression, and neutrophil transmigration. 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