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Preserved PC content coexisted with disturbed other lipids and organelles. The linked issue-wide erratum corrected publication year to 2020, not these biological findings (PMID 32333675; doi:10.1093/brain/awaa007).\nexposure: SLC44A1 frameshift genotypes, chronic choline treatment and acute iron challenge\nevidence_span: {\"source_cache\": \"artifacts/choline-research/31855247.abstract.txt\", \"locator\": \"Primary indexed abstract; zero-based, end-exclusive Unicode character offsets\", \"file_sha256\": \"34cbef18c0e83c0bcd06954a1d81d7329825171b5ecac31f8ea19b2457543f19\", \"start_char\": 0, \"end_char\": 1785, \"text_sha256\": \"34cbef18c0e83c0bcd06954a1d81d7329825171b5ecac31f8ea19b2457543f19\"}\n[choline-p31855247] Choline transporter-like 1 deficiency causes a new type of childhood-onset neurodegeneration. 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