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The 2016 correction changes the family A deletion in Figure 1 to exons 3 and 4; it is an author correction, not a scientific conflict.","comparator":null,"unit":null,"notes":"","entity":null},{"dimension":"nutrient_topic","value_text":"Manganese research collection; topical membership is not evidence of a direct dietary effect.","comparator":null,"unit":null,"notes":"","entity":{"slug":"manganese","display_name":"Manganese","entity_type_key":"nutrient_element"}},{"dimension":"organism","value_text":"Homo sapiens","comparator":null,"unit":null,"notes":"","entity":null},{"dimension":"plain_language","value_text":"Failure of manganese handling can produce excess manganese rather than shortage.","comparator":null,"unit":null,"notes":"","entity":null},{"dimension":"primary_references","value_text":"[mn-clin-tuschl2012] Syndrome of hepatic cirrhosis, dystonia, polycythemia, and hypermanganesemia caused by mutations in SLC30A10, a manganese transporter in man. 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The 2016 correction changes the family A deletion in Figure 1 to exons 3 and 4; it is an author correction, not a scientific conflict.\nexposure: Homozygous SLC30A10 changes in affected individuals without environmental overexposure.\ncross_nutrient: Hepatic manganese accumulation (affected_site); Polycythemia (associated_endpoint); Manganese (accumulated_nutrient)\nevidence_span: {\"source_cache\": \"artifacts/manganese-clinical-sources/tuschl2012.abstract.txt\", \"locator\": \"Indexed primary abstract\", \"file_sha256\": \"2f9189a6c46181f8b859a3e9815440d331870e6e4d78035218de33c46fa92e49\", \"start_char\": 0, \"end_char\": 1389, \"text_sha256\": \"2f9189a6c46181f8b859a3e9815440d331870e6e4d78035218de33c46fa92e49\", \"text_characters\": 1389}\n[mn-clin-tuschl2012] Syndrome of hepatic cirrhosis, dystonia, polycythemia, and hypermanganesemia caused by mutations in SLC30A10, a manganese transporter in man. 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