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The indexed abstract does not specify every line, dose or treatment duration; rescue supports a homeostasis role rather than establishing a transport stoichiometry.\nexposure: Mn2+ supplementation of depleted cells.\ncross_nutrient: TMEM165 Golgi cation-homeostasis protein (affected_protein); Golgi apparatus (affected_compartment)\nevidence_span: {\"source_cache\": \"artifacts/manganese-glycosylation-sources/potelle2016.abstract.txt\", \"locator\": \"Indexed primary abstract\", \"file_sha256\": \"e1845bfbdbb15370fd1375534464f61aa6e5674cc4129b6f0992d9a022882cc3\", \"start_char\": 0, \"end_char\": 1190, \"text_sha256\": \"e1845bfbdbb15370fd1375534464f61aa6e5674cc4129b6f0992d9a022882cc3\", \"text_characters\": 1190}\n[mn-gly-potelle2016] Glycosylation abnormalities in Gdt1p/TMEM165 deficient cells result from a defect in Golgi manganese homeostasis. 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