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A rat model of the disease.\nexposure: Chronic alcohol feeding with zinc status measured\nevidence_span: {\"source_cache\": \"artifacts/alcohol-research/26585415.abstract.txt\", \"locator\": \"Indexed abstract; zero-based, end-exclusive Unicode character offsets\", \"file_sha256\": \"6743a8cefec8f9b3504ea8cc068c50c2b8fca42934fef4309d7e9819ff10d28c\", \"start_char\": 0, \"end_char\": 1907, \"text_sha256\": \"6743a8cefec8f9b3504ea8cc068c50c2b8fca42934fef4309d7e9819ff10d28c\"}\n[alcohol-p26585415] Defect of mitochondrial respiratory chain is a mechanism of ROS overproduction in a rat model of alcoholic liver disease: role of zinc deficiency. (2016). https://pubmed.ncbi.nlm.nih.gov/26585415/ DOI: 10.1152/ajpgi.00270.2015","model_system":"Rat model of alcoholic liver disease with mitochondrial respiratory chain assays","directness":"author_interpretation","verification_status":"source_derived_draft","notes":"Exact curation-document quotation, not publisher quotation. 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